Why do mitochondria come up so often in conversations about energy and longevity, but rarely when women are trying to understand what is happening to their bodies in menopause?
Most people have heard mitochondria described as the “powerhouse of the cell.” But they also play a role in muscle function, recovery, cellular communication, and hormone production. As we age, damaged mitochondria can build up while the body becomes less efficient at clearing them away.
Could some of the fatigue, brain fog, reduced strength, and poor recovery women are told to accept as part of menopause or aging also be connected to declining mitochondrial health?
Perimenopause dietitian-nutritionist, Jen Scheinman, explains why mitochondria deserve a bigger place in the menopause conversation.
We explore the connection between estrogen and mitochondrial function, how mitophagy helps remove damaged mitochondria, and how urolithin A may support that process.
What You'll Discover In This Episode
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Why mitochondria are often overlooked in menopause conversations
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What mitochondria do beyond producing energy
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How mitochondrial decline may contribute to fatigue and poor recovery
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The connection between estrogen and mitochondrial function
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What mitophagy is and why it slows with age
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How urolithin A supports mitochondrial cleanup
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How urolithin A differs from CoQ10 and NAD boosters
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What the research suggests about strength, endurance, and recovery
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Why mitochondrial support cannot replace sleep, exercise, and nutrition
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